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The increase in antioxidant defense was supported by the reduced ROS levels observed in 1919 GBM PSPH amp and 1919 GBM PSPH V116I cells compared to parental cells (Fig

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This structural modification increases its stability and resistance to rapid enzymatic degradation in test models

Currently, therapeutic options for metastatic bladder cancer remain limited, highlighting the urgent need for novel treatment strategies and a better understanding of the molecular mechanisms underlying this disease 5,6,7

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